2005

Pathogenesis Of Cortical Lesions In A Model Of Tuberous Sclerosis

Arnold R. Kriegstein, MD, PhD
University of California, San Francisco
 

Sixty to ninety percent of patients with tuberous sclerosis have difficulty controlling epilepsy due to genetically abnormal progenitor cells, which hamper proper cortical development. In this study, observation of the proliferation and migration of these cells will add valuable insight into how these gene defects influence cortical development and on potential new therapies.

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More Research in the Area of Genetics

Gene Therapy for the Catastrophic Type 1 Progressive Myoclonus Epilepsy (EPM1)

Berge Minassian, MD

The University of Texas Southwestern Medical Center

The Joseph Gomoll Foundation
Tribute

Convergence of High Frequency Oscillations with Genetic and Epigenetic Profiles in Epileptic Networks: Towards Novel Diagnostics in Focal Epilepsies

Gemma Carvill, PhD

Northwestern University

Multimodal Profiling of Neuronal Morphology, Physiology and Gene Expression in Focal Cortical Dysplasia

Cathryn Cadwell MD, PhD

University of California, San Francisco

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